Early Evolution of Hepatitis C Virus (HCV) Quasispecies after Liver Transplant for HCV‐Related Disease
Evelyne Schvoerer
(1, 2)
,
Eric Soulier
(2)
,
Cathy Royer
(2)
,
Anne-Catherine Renaudin
(1)
,
Christine Thumann
(2)
,
Samira Fafi-Kremer
(2, 1)
,
Nicolas Brignon
(2)
,
Nicolas Meyer
,
Patricia Pinson
(3)
,
Bernard Ellero
(4)
,
Marie-Lorraine Woehl-Jaegle
(5)
,
Carole Meyer
(5)
,
Philippe Wolf
(5)
,
Pierre Zachary
(1)
,
Thomas F. Baumert
(2)
,
Françoise Stoll-Keller
(2, 1)
Evelyne Schvoerer
- Fonction : Auteur
- PersonId : 172853
- IdHAL : evelyne-schvoerer
- ORCID : 0000-0003-0290-4532
- IdRef : 083596917
Samira Fafi-Kremer
- Fonction : Auteur
- PersonId : 1062952
- ORCID : 0000-0003-3886-7833
- IdRef : 094693501
Nicolas Meyer
- Fonction : Auteur
Thomas F. Baumert
- Fonction : Auteur
- PersonId : 740312
- IdHAL : thomas-f-baumert
- IdRef : 143387898
Résumé
End-stage liver disease as a result of chronic hepatitis C virus (HCV) infection is the main indication for liver transplant (LT), but allografts are systematically infected with HCV soon after transplant. Viral quasispecies are poorly described during the early posttransplant period.